History
A 1987 study showed sugar acted as an analgesic drug whose effects could be blocked by a morphine blocker. In her 1998 book, author Kathleen DesMaisons outlined the concept of sugar addiction as a measurable physiological state caused by activation of opioid receptors in the brain and hypothesized that dependence on sugar followed the same track outlined in the DSM IV for other drugs of abuse.
Fifteen years later, 2002 research at Princeton began showing the neurochemical effects of sugar, noting that sugar might serve as a gateway drug for other drugs. The research group fed chow to the rats as well as a 25% sugar solution similar to the sugar concentration of soft drinks. After one month the rats became "dependent" on the sugar solution, ate less chow and increased their intake of the sugary drink to 200%. The sugar industry asserts that similar effects have been reported for rats given solutions that tasted sweet, but contained no calories. However, some scientists say that caloric value may not be the issue. Researchers say that sugar and the taste of sweet is said to stimulate the brain by activating beta endorphin receptor sites, the same chemicals activated in the brain by the ingestion of heroin and morphine.
In 2003, a report commissioned by two U.N. agencies at the World Health Organization and the Food and Agriculture Organization was compiled by a panel of 30 international experts. It recommended that sugar not account for more than 10% of a person's diet. However, the U.S. Sugar Association asserted that other evidence indicates that a quarter of our food and drink intake can safely consist of sugar.
Finally, a 2008 study noted that sugar affects opioids and dopamine in the brain, and thus might be expected to have addictive potential. It referenced bingeing, withdrawal, craving and cross-sensitization, and gave each of them operational definitions in order to demonstrate behaviorally that sugar bingeing is a reinforcer. These behaviors were said to be related to neurochemical changes in the brain that also occur during addiction to drugs. Neural adaptations included changes in dopamine and opioid receptor binding, enkephalin mRNA expression and dopamine and acetylcholine release in the nucleus accumbens.
Read more about this topic: Sugar Addiction
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